The same brain that is READING the Planck length is RENDERING the Planck length. I believe the potential for “looping” implicit in this arrangement may be at the core not only of diseases of the central nervous system, such as Parkinson’s and Lou Gehrig’s disease, but also cancer, Autism, and ME/CFS.
In this model, time writ large is an explosion (the Big Bang) that has layered membranes or branes, like an onion. These membranes, each representing a factor of light’s speed, are pierced by an individual arrow of time—time writ small. The point of intersection of time’s arrow with a brane of light’s speed will be treated here as a 2D hologram.
For any given point, a corresponding point on the brane behind it will appear supersaturated (e.g. sun); and a corresponding point on the brane in front of it will appear dilated (e.g. moon).
If I mis-read the Planck length as longer than it is, as if observing the holographic image from behind, the plain light of day may appear dilated to me. In response, my brain may try to slow time down, perhaps with dopamine. Now I get caught in a loop. The more I slow time down, the more I think the Planck length is longer than it is, so I slow time down again. Could this play a role in the core etiology of Parkinson’s?
If I mis-read the Planck length as shorter than it is, as if observing the holographic image from in front, the plain light of day may appear supersaturated to me. In response, my brain may try to speed time up, perhaps with serotonin. Now, too, I get caught in a loop. The more I speed time up, the more I perceive the Planck length as shorter than it is, so I speed time up again. Might this play a role in the core etiology of Stephen Hawking’s and Lou Gehrig’s disease (ALS)?
In Parkinson’s, if I am viewing the cone of time from its pit, my brain may use too much extracellular potassium. Once I am using too much extracellular potassium, it reinforces the original problem. If I read the world as having a condensing force, it forces me to remain at the base of time’s cone, where there is adequate exploding force to balance it.
In ALS, if I am viewing the cone of time from its mouth, my brain may use too much intracellular sodium. Once I am using too much intracellular sodium, it reinforces the original problem. If I view the world as having an exploding force, it forces me to remain at the mouth of time’s cone, where there is adequate collapsing force to oppose it.
Is it truly intracellular sodium, or is it just water that reads as sodium against a potassium background?
Is it truly extracellular potassium, or is it just water that reads as potassium against a sodium background?
The same thing, e.g. a vertical column of water—a fountain—can be perceived as having the exploding force or the collapsing force, depending on the orientation of the observer. The water in the middle of a fountain is both shooting up and falling down.
When the body is at the pit of time’s cone, is it because the observer (the pineal gland) is too high energy (too dilated, too watery)? Are we seeing a human body “at the pit of time’s cone” in the Atacama skeleton?
When the body is at the mouth of time’s cone, is it because the observer (the pineal gland) is too too low energy (not dilated enough, the crystal is too dense)?
The cone of time:
Dante’s Inferno Image: Galileo Galilei. Dante Collection, LITS, Mount Holyoke College
Atacama Skeleton. Photograph: Dr Emery Smith
More at “Perception, the Key to Disease.” https://www.hormonesmatter.com/perception-the-key-to-disease/
